There was an issue during submission. How is lichen sclerosus treated? Citations Publications citing this paper. Create a personal account to register for email alerts with links to free full-text articles. Treat with long-term, high-potency topical corticosteroids, close monitoring, and sexual and psychologic support.
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Manifestations of Lichen Sclerosus

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Topical tacrolimus for recalcitrant vulvar lichen sclerosus. Remarkably, none of the 11 samples was positive for Borrelia by PCR, while 6 of 10 out of the same 11 cases were positive by FFM [ 45 ], indicating the difficulties of the various techniques to reliably detect Borrelia in tissue specimens. Lichen sclerosus in Adult. Topical FK suppresses cytokine and costimulatory molecule expression in epidermal and local draining lymph node cells during primary skin immune responses. Long-term follow-up and ongoing suppressive treatment are mandatory. Vulvar LS often begins around the periclitoral hood with a sharply demarcated, slightly elevated, nonspecific erythema Fig. One-third reported a transient, mild, local burning sensation.
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Lichen sclerosus

Clinical and histologic effects of topical treatments of vulval lichen sclerosus: It's not thought to be possible to prevent vulval cancer completely, but you may be able to reduce your risk by:. Topical Calcineurin Inhibitors The topical calcineurin inhibitors TCIs pimecrolimus and tacrolimus have a significant anti-inflammatory activity, immunomodulatory effects, and a low systemic immunosuppressive potential. The urethral meatus finally exposed behind the previously fused labia minora thin arrow.. The etiology of LS has not yet been adequately explained, but there is increasing evidence that autoimmune mechanisms play a pathogenetic role.
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The treatment of balanitis xerotica obliterans. Topical Calcineurin Inhibitors The topical calcineurin inhibitors TCIs pimecrolimus and tacrolimus have a significant anti-inflammatory activity, immunomodulatory effects, and a low systemic immunosuppressive potential. The labia minora may become reabsorbed and fused, and the clitoris entrapped under scar tissue. IPP with clinical and histologic features of LS has been described. There is evidence supporting an autoimmune etiology in genetically predisposed patients. An individual case of extragenital LS treated successfully with narrow-band UVB has been reported [ ].
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